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Feed Plant Chemical Additive Service Provider, Zhuhai City, Zhuhai (2026)

08/04/2026

Recently, the price of vitamin E has skyrocketed. With such high prices, we are still using it in our feed. So, where is the value of vitamin E?
Vitamin E is the "secret weapon" for improving pork quality! As a powerful fat-soluble antioxidant, vitamin E protects unsaturated fatty acids in muscle cell membranes from oxidation, reducing drip loss and extending shelf life. Studies show that adequate vitamin E in diets (recommended 50-100 IU/kg for finishing pigs) reduces drip loss by 20-30%, gives redder meat color, and extends shelf life by 3-5 days. Vitamin E also inhibits off-flavors from lipid oxidation, preserving natural pork flavor. For farms premium pork, vitamin E is indispensable. Good meat commands good price – start with vitamin E!

Mechanisms of vitamin E improving pork quality: First, antioxidant protection: vitamin E is the primary antioxidant in cell membranes, scavenging free radicals, preventing phospholipid peroxidation, maintaining membrane integrity, reducing drip loss. Second, stabilizing meat color: protects myoglobin from oxidation, maintaining color, delaying discoloration. Third, inhibiting off-flavors: reduces aldehydes, ketones from lipid oxidation, preserving natural pork flavor. Fourth, extending shelf life: delays microbial growth, extending chilled meat shelf life by 3-5 days. Fifth, improving tenderness: protects muscle fiber structure, reducing protein oxidation, maintaining tenderness. Recommended levels: finishers 50-100 IU/kg, supplementation 4-6 weeks pre-slaughter maximizes meat quality improvement.

03/03/2026

Vitamin K3 is a core nutrient for maintaining normal blood clotting function in poultry, playing an indispensable coenzyme role in the liver's synthesis of prothrombin and clotting factors II, VII, IX, and X. It mediates the carboxylation of glutamate residues in the precursors of these factors, ensuring the smooth progress of the calcium-mediated coagulation cascade, thereby protecting vascular integrity and preventing bleeding tendencies.

When dietary vitamin K3 supply is insufficient or absorption is impaired, it directly leads to the inhibition of the synthesis of these clotting factors, prolonging clotting time. In chicks, clinical manifestations include widespread subcutaneous hemorrhage, intramuscular hemorrhage, and coelomic hematomas; in severe cases, acute death can result from internal bleeding. In laying hens, increased fragility of oviduct microvessels leads to bleeding during egg production, blood spots on the eggshell surface, and a significant reduction in egg quality. In modern high-density farming, subclinical coccidiosis infection and the presence of mycotoxins in feed often further exacerbate vitamin K depletion and absorption disorders.

Precise nutritional supply is key to preventing these problems. Based on the National Nutrition Requirements for Poultry (NRC) and DSM's OVN (Optimal Vitamin Nutrition Standard), it is recommended that the amount of vitamin K3 added to the diet should be precisely rationed according to breed and production stage: 2-4 mg/kg of complete feed is recommended for chicks to meet the needs of rapid growth and tissue development; 2-3 mg/kg is recommended for laying hens to maintain the health of the reproductive tract mucosa; and 3-5 mg/kg is recommended for breeder hens to ensure fertilization rate of hatching eggs and normal embryonic development. Studies have shown that maintaining adequate and stable vitamin K3 levels in the diet can reduce the overall incidence of clinical hemorrhagic diseases in poultry by 80-90%, while significantly reducing the proportion of blood-spotted eggs and improving the commercial value of eggs.

Given that vitamin K3 is a fat-soluble vitamin, and that ordinary premixed formulations are prone to potency degradation during high-temperature granulation and long-term storage, it is recommended to use a coating or sodium menadione bisulfite complex with good stability to ensure that the actual intake by poultry meets physiological needs, achieving a dual improvement in efficient farming and animal welfare.
K3 nutrition feed

26/02/2026

Vitamin B1 is the "starter" of appetite! As a key coenzyme in energy metabolism, vitamin B1 participates in converting carbohydrates to energy, directly affecting feeding center function. Vitamin B1 deficiency causes reduced appetite, decreased feed intake, growth retardation. Studies show adequate vitamin B1 in diets (recommended: piglets 2-4 mg/kg, growers 2-3 mg/kg) increases feed intake by 10-15%, promoting growth. Keep your herd's appetite strong – start with vitamin B1!

22/02/2026

Free Radicals and Vitamin E: The Mechanism of Action of the Antioxidant Defense Line
Keywords: Free radicals, oxidative stress, vitamin E, antioxidants, lipid peroxidation, cell protection
I. The Dual Role of Free Radicals
Free radicals are natural byproducts produced during normal metabolism in organisms. Within appropriate ranges, they play a positive defense and protective role in the body, such as participating in immune responses and helping to reduce the attack of bacteria and parasites on the body. However, when free radicals accumulate excessively, their destructive effects become apparent.
II. Potential Hazards of Free Radicals
Excessive free radicals can trigger a series of chain reactions:
Catalyzing lipid peroxidation: Attacking polyunsaturated fatty acids in cell membranes, disrupting membrane integrity and fluidity.
Damaging enzyme activity: Oxidizing and modifying key enzyme proteins, leading to metabolic disorders.
Damaging organelles: Damaging unstable organelles such as mitochondria and endoplasmic reticulum, affecting normal cell function.
Altering membrane structure:Ultimately leading to irreversible damage to cells and subcellular structures.
III. Antioxidant Protection Mechanism of Vitamin E
Vitamin E, as an important lipid-soluble intracellular antioxidant, plays a crucial role in defending against free radical damage:
Stabilizing ingested polyunsaturated fatty acids, preventing excessive oxidation in the body.
Clearing intermediate metabolites produced during lipid synthesis and degradation.
Terminating the chain reaction of lipid peroxidation, protecting the structural integrity of cell membranes and subcellular organelles.
IV. Summary
Free radicals are a double-edged sword in the body's metabolism—in appropriate amounts, they are defensive guardians; in excess, they are destructive factors. Vitamin E, through precise antioxidant regulation, establishes a balance between the production and scavenging of free radicals, protecting cells from oxidative damage and serving as an indispensable nutritional defense line for healthy animal husbandry.

21/02/2026

Vitamin E and Immunity: An Excellent Immune Enhancer

Keywords: Vitamin E, immune adjuvant, antioxidant, free radical scavenging, lymphocyte proliferation, disease resistance

I. Vitamin E: An Excellent Immune Enhancer

Vitamin E is a recognized excellent immune enhancer, often used as an immune adjuvant in animal nutrition. Supplementing with high levels of vitamin E can:

Increase antibody levels, enhance humoral immunity, strengthen phagocytic activity, and enhance non-specific immunity

Upregulate cytokine levels related to the immune response, coordinating immune responses

II. Classic Studies

1. Enhancing Immune Organ and Cell Activity

In 1974, Tengerdy's research found that after vitamin E administration, the number of antibody-producing cells in animals significantly increased, spleen weight increased, and the number of macrophages in the reticuloendothelial system increased, indicating that vitamin E enhances the body's defense capabilities at the level of immune organs.

2. Enhancing Anti-Infection Capacity

Tengerdy et al. (1975) further found that high doses of vitamin E can effectively protect chickens against E. coli infection; this protective effect is closely related to enhanced phagocytic cell activity and an increased number of antibody-forming cells.

3. Promoting Lymphocyte Proliferation
Bendich (1986), Bonneffe (1988), Kramer (1991), and others have demonstrated that vitamin E supplementation can promote lymphocyte proliferation in different animals, enhance cellular immune function, and provide strong support for vaccine immunization effects.

III. The Balance Between Vitamin E and Free Radicals
The Dual Role of Free Radicals
Free radicals are normal metabolic products of organisms. In appropriate amounts, they can play a protective role in the body, helping to reduce the attack of bacteria and parasites. However, excessive free radicals will:
catalyze lipid peroxidation reactions, damaging cell membrane structure
damage enzyme activity and unstable intracellular organelles
lead to damage to cell structure and function
The Antioxidant Protection Mechanism of Vitamin E
As an intracellular fat-soluble antioxidant, vitamin E can:
stabilize ingested polyunsaturated fatty acids, preventing their excessive oxidation in the body
clear intermediate metabolites produced during lipid synthesis and degradation
protect the structural integrity of cell membranes and subcellular organelles (such as mitochondria and endoplasmic reticulum)
Summary
Vitamin E comprehensively enhances animal disease resistance by strengthening the function of immune organs, promoting antibody production, and activating immune cells. Meanwhile, its powerful antioxidant properties effectively scavenge excess free radicals and protect immune cells from oxidative damage. In the context of antibiotic-free farming, vitamin E, as a safe and highly effective immune enhancer, deserves significant attention in dietary intake.

20/02/2026

Possible Mechanisms of Vitamin A and β-Carotene in Enhancing Disease Resistance

Keywords: Vitamin A, β-carotene, immune regulation, epithelial barrier, antibody synthesis, antioxidant, steroid hormones

I. Maintaining Epithelial Barrier Integrity, Building the First Line of Defense
Vitamin A participates in the differentiation and maintenance of epithelial cells, preventing keratinization of mucosal epithelium and ensuring the integrity of mucosal barriers in the respiratory, digestive, and urinary/reproductive tracts, thereby effectively preventing pathogen invasion and protecting animals from infection.

II. Promoting Antibody Synthesis and Enhancing Humoral Immune Response
Vitamin A directly participates in the synthesis of immunoglobulins (such as IgG and IgA), enhancing antibody responses to antigens. β-carotene can also indirectly support this process through metabolic transformation, jointly enhancing the body's specific immune function.

III. Exercising Antioxidant Properties and Scavenging Free Radical Damage
Both vitamin A and β-carotene have significant antioxidant capabilities, regulating the concentration of highly reactive oxygen molecules (such as free radicals) within cells, protecting the structural and functional integrity of immune cell membranes, and mitigating the inhibitory effect of oxidative stress on the immune system.

IV. Steroid Hormone-like Functions, Regulating Immune Cell Activity
Vitamin A and its metabolites (such as retinoic acid) have steroid hormone-like regulatory effects, influencing the differentiation and function of immune cells (such as T cells, B cells, and macrophages), and regulating the intensity and direction of the immune response at the gene expression level.

19/02/2026

Nutritional Immunity: The Fundamental Path to Animal Health

Keywords: Immune system, nutritional immunity, vitamins, disease resistance, antibiotic-free farming

I. The Core Value of the Immune System
Strengthening the immune system is the fundamental way to keep animals healthy and improve their productivity. The immune system plays three core roles:

Resistance: Resisting the invasion of pathogens such as bacteria, viruses, fungi, and parasites.

Elimination: Clearing invading pathogens and their toxins.

Repair: Repairing damaged tissues and restoring normal bodily functions.

A weakened immune system directly reduces an animal's resistance to disease, leading to illness. A healthy immune system is more effective than any drug—it resists external disease invasion without any drug side effects.

II. The Synergistic Relationship Between Nutrition and Immunity
An animal's nutritional status is closely related to its immune function, and the two interact:

Nutrition Affects Disease Manifestations: Determining the pathogenicity, frequency, and duration of disease.

Nutrition Regulates Immune Capacity: Key nutrients directly affect the activity of immune cells and antibody production.

Immune Response Increases Nutritional Needs: When animals are under immune stress, their need for specific nutrients increases significantly.

III. Vitamins: The Key Support for the Immune System
Adequate dietary vitamins are crucial for maintaining a strong immune system. Studies have shown that several vitamins play an irreplaceable role in enhancing animal immunity:

Vitamin A: Maintains the integrity of mucosal barriers, building the first line of defense.

Vitamin E: A powerful antioxidant, protecting the structure of immune cell membranes.

Vitamin C: Anti-stress, promoting antibody production.

B vitamins: Participate in energy metabolism, supporting immune cell proliferation.

It is worth noting that the vitamin levels required to support optimal immune function are often higher than the requirements for normal growth and reproduction. This means that during critical stages (such as weaning, regrouping, and periods of high disease incidence), targeted vitamin supplementation is necessary.

Choosing a scientific nutritional plan is choosing the most powerful "invisible vaccine." Let's start with nutritional immunity to build a solid barrier for animal health!
# Vitamins # Feed # Feed Additives # Breeding # Suppliers

13/02/2026

Additional biotin must be added to the feed (corn soybean meal type diet has insufficient content).
Reproductive performance (weaned piglets, hooves).
The production performance of pigs (hooves, growth rate, fat composition).
Production performance of laying hens (fatty liver, fertilization rate, protein quality).
Production performance of broiler chickens (FLKS, SDS, claws).
Cow production performance (hooves, milk yield).
The function of biotin in intermediate metabolism is related to a series of carboxylation reactions.
The acetyl CoA carboxylation reaction is a key reaction for the synthesis of fatty acids in vivo, and this carboxylation reaction relies on the participation of biotin;
The carboxylation of pyruvate to oxaloacetate to produce sugars (gluconeogenesis) is a necessary pathway for many non sugar substances to produce sugars, which relies on biotin;
Biotin plays an important role in gluconeogenesis. When carbohydrate intake is insufficient, glucose is generated from fat and protein through gluconeogenesis to maintain blood glucose concentration;
Lack of biotin leads to insufficient formation of oxaloacetate and succinic acid, resulting in a decrease in the amount of amino acids incorporated into proteins. # Vitamins # Feed # Feed Additives # Breeding # Suppliers

12/02/2026

The application of vitamins in feed
B6 pyridoxine
Mechanism of action:
It is a coenzyme involved in protein and ammonia metabolism. When insufficient, the activity of amino acid transferase decreases, thereby reducing the efficiency of amino acid synthesis of proteins.
Performance related to growth:
Significant impact on food intake
Save methionine
Improve protein deposition
, acid, additive, additive

11/02/2026

The impact of biotin on cattle farming
1. Unreasonable dietary combinations result in high protein content and imbalanced amino acid ratios, requiring biotin for the breakdown and metabolism of excess amino acids (especially branched chain amino acids, leucine, and isoleucine);
2. Certain diseases and functional disorders hinder the absorption of biotin;
3. Using feed with low biotin content such as grain by-products as the main component of the diet, while reducing the use of feed rich in biotin (such as whey, fermentation by-products, yeast, and alfalfa powder);
4. The imbalance of the ratio of fine to coarse grains leads to excessive acidity in the rumen, resulting in the destruction of biotin;
5. The use of antibiotics in feed inhibits the activity of bacteria in the rumen and intestines, leading to obstacles in biotin synthesis;
6. After the feed becomes moldy and oxidized, the biotin antagonists produced by the mold will reduce the utilization rate of biotin;
7. Long storage time of feed, high temperature, high humidity, etc. can all cause loss of biotin;
8. Calves are prone to biotin deficiency due to incomplete rumen development and incomplete synthesis function;

Feed Plant Chemical Additive Service Provider

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